By Kenneth J. Zucker (ed.)

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Extra resources for Archives of Sexual Behavior, Vol 39, issue 2, April 2010

Example text

In an early study, Morokoff and Heiman (1980) found no significant differences in VPA between women diagnosed with sexual arousal disorder and a control group of women. In a study of premenopausal women with sexual arousal problems, following suggested definitions of Basson et al. (2003), women were classified into three subtypes: genital, subjective, and combined (subjective and genital) sexual arousal disorder (Brotto, Basson, & Gorzalka, 2004). Only those women in the ‘‘genital’’ subgroup, characterized by self-reports of impaired genital sensitivity, showed evidence of impaired genital response.

The relationship between distress and symptoms of FSAD will be discussed below. ’’ In DSM-III-R (American Psychiatric Association, 1987), subtyping (lifelong or acquired; generalized or situational; psychogenic only or psychogenic and biogenic) had been added. , ED) (Segraves, 1996b), the lack of empirical data on the relationship between severity and duration criteria and treatment outcome ruled this out. For FSAD, however, the DSM-IV text included this statement: ‘‘Occasional problems with sexual arousal that are not persistent or recurrent…are not considered to be Female Sexual Arousal Disorder’’ (p.

Sexual ‘‘desire’’ may reflect early arousal processes (Everaerd, Laan, Both, & van der Velde, 2000) and it is argued that there is no such thing as spontaneous sexual desire (Laan & Both, 2008). Sexual thoughts or sexual activity act as stimuli, which then trigger the desire-arousal process. Individuals have variable tendencies to respond to sexual stimuli (often referred to as ‘‘arousability’’) (Laan & Both, 2008). While the drive model assumes that we have sex because we feel desire (Laan & Janssen, 2007), the incentive motivation model instead suggests that we feel sexual desire because we have sex or think about sex (Laan & Both, 2008).

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