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34] Grof P. Excellent lithium responders: people whose lives have been changed by lithium prophylaxis. In: Birch NJ, Gallicchio VS, Becker RW ed. Lithium: 50 Years of Psychopharmacology, New Perspectives in Biomedical and Clinical Research. Cheshire, Connecticut, Weidner Publishing Group, 1999:36-51. [35] Guscott R, Taylor L. Lithium prophylaxis in recurrent affective illness. Efficacy, effectiveness and efficiency. Br J Psychiatry 1994, 164, 741-746. [36] Haas M, Schooler J, Tosteson DC. Coupling of lithium to sodium transport in human red cells.

Patients on lithium and antidepressants were matched according to gender (21 male and 31 female in each group), age, and duration of antidepressant treatment (mean 5 years in both groups). The percentage of recurrent labial herpes in all groups ranged between 38% and 48%. The assessment of recurrence rates for labial herpes infections before and during pharmacological treatment made in Polish patients showed that among 28 patients with recurrent herpes before lithium treatment, in 13 patients (46%), a total disappearance of recurrences was observed, in seven the frequency diminished, in six remained the same and in two it increased.

It was demonstrated that moodstabilizing drugs and lithium in particular may act to prevent neuronal damage and tissue loss that occur in the brain of patients with bipolar disorders. Lithium has been shown to produce neuroprotective effects by multiple mechanisms. One target of such effect has been the brain-derived neurotrophic factor (BDNF), an important modulator of neuronal proliferation and synaptic plasticity. Lithium administration induces BDNF and its receptor in frontal cortex and hippocampus of experimental animals [26, 38].

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