By Sudarshan K. Malhotra
A part of a sequence which goals to hide contemporary wisdom within the box of neural technological know-how, this quantity discusses such issues as: the molecular bases of nerve regeneration; plasticity of descending spinal pathways in constructing mammals; and improvement of the mammalian auditory hindbrain.
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Extra info for Advances in Neural Science, Volume 2 (Advances in Neural Science)
Again, the physiological significance of the transient expression of SRIF receptors in the cerebellum can only be speculated on at present. At the microscopic level, SRIF receptors are associated with the external granule layer and thus may play a role in the proliferation of the stem cells of the granule cells, and their disappearance appears to coincide with the migration of the neuroblasts. The hypothesis that SRIF may act as atrophic factor involved in the regulation of cell division and/or migration in the external granular layer is supported by the absence of synaptic contacts in this layer, thus suggesting that SRIF receptors in this layer are not involved in neurotransmission.
1986). A decrease in this particular class of steroid (after adrenalectomy) appears to increase the potential for altering the expression of the propressophysin gene within this nucleus. , 1988) found that adrenalectomyinduced plasticity in VP expression is further enhanced by ablation of a specific afferent pathway. The authors suggest that synaptic events may interact with the hormonal environment to regulate neuronal phenotypes. D. Vasoactive Intestinal Polypeptide Using dissociated cultures of embryonic rat spinal cord and dorsal root ganglia, Eiden et al.
They examined the glial scar in the transected optic nerve of the albino rabbit by quantitative receptor autoradiography. Ligand displacement and saturation experiments indicate that SP and receptor binding sites expressed by the glial scar have pharmacological characteristics similar to those of SP receptors in the rabbit striatum, rat brain, and rat and canine gut. It is suggested that SP may play a role in regulating the glial response to injury in the CNS. , 1988). D. , 1987), very little is known ontogenetically.